Synopsis
Recognise systemic venous congestion and acute RV shock, identify the cause and balance preload, afterload and contractility without reflex fluid or vasodilator harm.
- Right-sided HF causes raised systemic venous pressure: raised JVP, peripheral oedema, ascites, hepatomegaly, early satiety and congestive renal/liver dysfunction.
- Common drivers are left-sided HF, pulmonary hypertension or lung disease, acute PE, RV infarction, tricuspid/pulmonary valve disease, cardiomyopathy and constrictive pericardial disease.
- Acute RV failure can present with shock and clear lungs; do not exclude cardiac shock because crackles are absent.
- Treat the cause while balancing RV preload: severe underfilling may justify a small monitored fluid challenge, but routine repeated boluses worsen dilation and septal shift when congestion is present.
- Use loop diuretic for systemic congestion, watching renal function and perfusion; venous congestion itself can drive cardiorenal dysfunction.
- Avoid nitrates and other preload-reducing drugs in hypotensive RV infarction; restore coronary flow urgently through the ACS pathway.
- Pulmonary vasodilators are disease-specific specialist therapy, not empirical treatment for undifferentiated RV failure and can be harmful in some left-heart or lung-disease phenotypes.
- Refractory shock needs expert vasoactive therapy and early mechanical-support/advanced-centre discussion.