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Educational draft · awaiting clinical reviewUse Rapid for revision, not patient-care decisions. Check current national and local guidance and the BNF or BNFC before acting.
RapidMLA

Primary and secondary immunodeficiency

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Synopsis

Recognise clinically important immune failure, distinguish inherited from acquired causes, map infection patterns to immune compartments and investigate urgently without delaying treatment of active infection.

  • Suspect immunodeficiency when infections are unusually severe, persistent, recurrent, treatment-resistant or caused by unusual organisms, rather than counting minor infections alone.
  • Primary immune regulatory disorders can present from infancy to adulthood and may cause autoimmunity, inflammation, lymphoproliferation or malignancy as well as infection.
  • Secondary causes are more common: medicines, haematological malignancy, HIV, protein loss, malnutrition, asplenia, renal disease and critical illness can impair different compartments.

Key red flags

Serious, persistent or unusual infection

Invasive infection, repeated hospitalisation, failure of standard treatment, opportunistic organisms or infection at multiple sites raises concern beyond ordinary frequency.

Early combined phenotype

Thrush, chronic diarrhoea, failure to thrive, persistent viral infection or vaccine-strain disease in infancy demands urgent specialist assessment.

Reasoning priorities

01
Documented infection phenotype

Retrieve organisms, culture sites, imaging, antimicrobial courses, severity and response across records.

Repeated viral upper-respiratory symptoms differ from culture-proven pneumonia, opportunistic infection or invasive encapsulated bacterial disease.

Worked reasoning

Worked caseAdult recurrent pneumonia

A 38-year-old has three radiologically confirmed pneumonias in two years, chronic sinusitis and low globulin; there is no known childhood diagnosis.

  1. Assess current infection severity and treat promptly, while building a verified timeline of organisms, sites, imaging, antibiotic response and structural lung damage.
  2. Recognise an antibody-pattern phenotype that can present in adulthood, but review secondary causes including B-cell-active medicines, haematological disease, HIV risk and protein loss.
  3. Order initial full blood count with film, quantitative immunoglobulins, renal and liver profiles, albumin and consented HIV testing, and discuss early with clinical immunology before unstructured panels.
  4. Verify abnormalities when clinically appropriate, arrange specialist functional testing and pulmonary assessment, and base prophylaxis or replacement on diagnosis and infection outcomes rather than one concentration.
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Sources and review status5 sources · checked 7 Sept 2026 · clinical review pending
Sources

Sources and review status

National guidance is shown before implementation-dependent detail. Apply principles in context and verify current guidance when a decision affects care. Source check completed 7 Sept 2026; clinical approval remains outstanding.

Authoring stateRapid draftClinical stateAwaiting reviewJurisdictionUnited Kingdom