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Non-contrast CT in acute neurological disease

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Imaging must not interrupt neurological resuscitation

Falling consciousness, new focal deficit, seizure, unequal pupils or a sudden severe headache can represent haemorrhage, large-vessel stroke, hydrocephalus or herniation even before CT is available.

Action: Stabilise airway, breathing and circulation, record glucose and neurological findings, activate the relevant stroke, trauma or neurosurgical pathway, and obtain urgent non-contrast CT when transport is safe.

Synopsis

Use non-contrast head CT safely in acute neurological presentations, recognise major patterns and limitations, and continue urgent diagnostic pathways when a negative scan cannot exclude the suspected disease.

  • Non-contrast CT is fast and excellent for acute haemorrhage, hydrocephalus, skull injury and substantial mass effect; it is less sensitive for early ischaemia and some posterior-fossa lesions.
  • Review systematically: technical adequacy, extra-axial spaces, cisterns and sulci, ventricles, parenchymal symmetry and grey–white differentiation, posterior fossa, skull and extracranial tissues.
  • Fresh blood is usually hyperattenuating, but anaemia, very small volume, lesion age and artefact can alter conspicuity; describe compartment and mass effect before inferring cause.

Key red flags

A fall in GCS, progressive focal deficit, new seizure or pupil asymmetry requires immediate reassessment and urgent imaging or repeat imaging despite an earlier reassuring scan.

Thunderclap headache with neck stiffness, vomiting, collapse or exertional onset remains concerning for subarachnoid haemorrhage even with a normal examination.

Anticoagulant exposure, bleeding tendency and recent trauma lower the threshold for urgent CT but do not identify the lesion or replace drug-specific haemostasis planning.

A normal early CT does not exclude acute ischaemia; for suspected SAH, a negative radiologist-reported CT within six hours may avoid routine LP, while a later negative scan can require LP at least 12 hours after onset.

Posterior-fossa and skull-base disease may be obscured by beam-hardening artefact; persistent brainstem or cerebellar signs need a more sensitive pathway.

Acute haemorrhage

Search sulci, basal cisterns, fissures, ventricles, brain parenchyma and extra-axial spaces. State side, compartment, volume impression, ventricular extension and associated oedema or shift.

Early ischaemia

Compare hemispheres for grey–white loss, focal hypoattenuation, sulcal effacement and a dense artery. Small or very early infarcts may remain invisible, especially in the posterior fossa.

Mass effect

Assess sulci, ventricles, basal cisterns, midline position and herniation corridors. Millimetres of shift alone do not determine clinical severity; lesion location and trajectory matter.

Negative scan boundary

Define the disease, timing and image quality before calling CT reassuring. A negative result is meaningful only for the question and population in which its performance is known.

Investigation priorities

01
Non-contrast CT headFirst step

Identify acute blood, major mass effect, hydrocephalus, fracture and established infarction rapidly.

Management branches

Worked acute pathwayThunderclap headache with a negative scan

An adult presents after sudden maximal-at-onset headache and initial non-contrast CT shows no blood.

  1. Confirm exact onset, neurological state, scan timing and technical adequacy, while maintaining urgent SAH assessment.
  2. If CT was obtained and radiologist-reported within six hours, use the NG228 pathway rather than adding routine LP automatically.
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Sources and review status4 sources · checked 13 Sept 2026 · clinical review pending
Sources

Sources and review status

National guidance is shown before implementation-dependent detail. Apply principles in context and verify current guidance when a decision affects care. Source check completed 13 Sept 2026; clinical approval remains outstanding.

  • NICE NG128 stroke and TIA recommendationsPublished May 2019; acute stroke and ICH recommendations, including imaging sequence, read in the current body on 13 September 2026. Applies to people over 16 and does not define paediatric pathways. Applied specifically to Non-contrast CT in acute neurological disease.
  • NICE NG228 aneurysmal subarachnoid haemorrhage recommendationsPublished 23 November 2022; recommendations 1.1.1–1.1.23 read for CT, timing-dependent negative scans, lumbar puncture, spectrophotometry and specialist transfer. The six-hour route assumes accurate onset, adequate imaging and radiologist reporting. Applied specifically to Non-contrast CT in acute neurological disease.
  • NICE NG232 head injury recommendationsPublished 18 May 2023; recommendations on acute assessment, CT criteria, neurosurgical discussion and neurological deterioration read 13 September 2026. This source governs traumatic head injury across ages and was not generalised to spontaneous disease. Applied specifically to Non-contrast CT in acute neurological disease.
  • ENLS Intracranial Hypertension and Herniation Protocol version 6.0Version 6.0, updated September 2024; communication, diagnosis, Tier Zero and Tier One sections read. Multi-aetiology professional consensus for the first hours of an ICP crisis, not a UK national or disease-specific outcomes guideline. Applied specifically to Non-contrast CT in acute neurological disease.
Authoring stateRapid draftClinical stateAwaiting reviewJurisdictionUnited Kingdom