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RapidMLAMSRAGP

Calciphylaxis

Essential points for quick revision.

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Painful necrosis with infection risk

Rapidly progressive retiform necrosis, severe pain, fever, hypotension, spreading erythema, purulence, crepitus, wet gangrene or a deteriorating dialysis patient may have calciphylaxis with superimposed cellulitis, sepsis or necrotising infection, a major cause of early death.

Action: Admit for ABCDE and sepsis care, urgent nephrology, dermatology, tissue-viability, pain and surgical assessment, culture and image as appropriate and begin antimicrobials and source control when infection is credible without delaying calciphylaxis-directed management.

Synopsis

Recognise painful calcific arteriolopathy before black necrosis appears, distinguish it from infection and other retiform disease, and coordinate urgent renal, wound, analgesic, metabolic and infection management despite limited treatment evidence.

  • Calciphylaxis is calcific uraemic arteriolopathy: an uncommon, intensely painful occlusive small-artery disease seen mainly in advanced CKD and dialysis, although non-uraemic cases occur.
  • Deep burning pain and firm woody subcutaneous induration can precede visible change; lesions evolve through mottled livedoid or retiform purple-grey plaques to ulcer and black eschar.
  • On brown or black skin, early colour may be deep violet, grey-brown or subtle; exquisite focal pain, hardness, temperature and an angular vascular outline should trigger examination under good light and touch.

Key red flags

Pain out of proportion, rapidly enlarging livedoid or retiform plaques, black eschar, new systemic illness, crepitus, hypotension, exposed deep tissue, genital involvement, uncontrolled opioid-resistant pain or acute tissue ischaemia requires same-day multidisciplinary escalation.

Necrotic ulcer and eschar

A painful irregular ulcer exposes yellow-black fat and develops adherent black eschar, with a high risk of bacterial invasion and sepsis.

Investigation priorities

01
Clinical risk and full-skin assessmentFirst step

Recognise the syndrome early and document distribution before ulceration obscures the active edge.

Management branches

First-line recognition sequenceAct on pain and induration before eschar

A dialysis or high-risk patient develops new focal severe skin pain, hardness or livedoid change.

  1. Examine the complete painful region and all skin with consent, document depth, temperature, retiform outline and infection and assess ABCDE and limb perfusion.
  2. Contact nephrology and dermatology the same day and involve tissue viability and pain specialists before waiting for ulceration or a biopsy appointment.

Key medicines

Intravenous sodium thiosulfateA common off-label haemodialysis regimen is 25 g intravenously during the final hour of or immediately after each dialysis session three times weekly, using a lower starting dose when body size or tolerance requires it.
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Sources and review status3 sources · checked 27 Aug 2026 · clinical review pending
Sources

Sources and review status

National guidance is shown before implementation-dependent detail. Typical adult dose examples remain subject to patient factors, contraindications and the live BNF or specialist protocol. Source check completed 27 Aug 2026; clinical approval remains outstanding.

Authoring stateRapid draftClinical stateAwaiting reviewJurisdictionUnited Kingdom