Synopsis
Recognise physiological danger and time-critical surgical causes of acute abdominal pain, resuscitate in parallel with diagnosis, and choose examination and imaging that lead to a definitive disposition.
- Decide whether the patient is unstable before refining the pain history: record respiratory rate, oxygen saturation, pulse, blood pressure, temperature, consciousness, perfusion and urine output.
- Treat pain and nausea early while repeating examination; analgesia supports humane care and does not prevent competent recognition of peritonism or deterioration.
- Pain beginning diffusely then localising can reflect evolving parietal irritation, while abrupt maximal-at-onset pain raises vascular rupture, ischaemia, perforation, torsion or renal colic.
- Always ask about previous abdominal operations, hernias, anticoagulants, NSAIDs, alcohol, gallstones, vascular disease, pregnancy possibility and relevant gynaecological or testicular symptoms.
- Peritonism is a syndrome of involuntary guarding, percussion or cough tenderness and rigidity; an older, immunosuppressed or steroid-treated patient may have dangerous disease with muted signs.
- A normal lactate does not exclude early mesenteric ischaemia, and a raised lactate is not specific; clinical probability determines urgent CT angiography and surgical discussion.
- Use ultrasound for focused biliary, pelvic and aortic questions, contrast CT for many undifferentiated acute abdomens, and arterial-phase imaging when vascular pathology is suspected.
- Keep the patient fasting when surgery or procedural sedation is plausible, establish venous access, correct fluid and electrolyte deficits, and give antimicrobial treatment promptly when sepsis or perforation is suspected.
Key red flags
Sudden abdominal, flank or back pain with syncope, shock or a known aneurysm should trigger immediate vascular consideration. The classic pulsatile mass may be absent, and transient response to fluid does not remove rupture risk.
Severe pain disproportionate to early abdominal findings, atrial fibrillation, atherosclerosis, low-flow state or postprandial chronic symptoms raises bowel ischaemia. Peritonism indicates advanced injury; normal early lactate cannot safely exclude it.
Abrupt severe pain, generalised guarding or rigidity, sepsis and free intraperitoneal gas support perforation. Absence of visible free gas on plain radiography does not exclude a contained or early perforation.
Colicky pain, vomiting, distension and obstipation suggest obstruction; constant worsening pain, focal tenderness, fever, tachycardia, acidosis or a closed-loop configuration raises compromised blood supply and demands urgent surgery.
Investigation priorities
Detect shock, sepsis, respiratory compromise and trajectory while diagnostic work continues.
Management branches
Abdominal pain accompanies shock, peritonism or another immediate physiological threat.
- Begin ABCDE, obtain large-bore venous access, send urgent bloods and group-and-save or crossmatch, monitor urine output and provide appropriate oxygen and warmed fluid or blood support.
- Call the relevant senior surgeon immediately and involve vascular, gynaecology, urology, anaesthesia or critical care according to the leading threat.
- Give analgesia and antiemetic treatment, start locally recommended antimicrobials when sepsis or perforation is suspected, and keep the patient fasting.
- Use only imaging that changes immediate definitive management; do not transfer an unstable patient to an unsuitable scanner without senior planning and resuscitation capability.
The patient is physiologically stable but pain is severe, focal or unexplained.
- Take a time-ordered history including operations, vascular, drug, alcohol, pregnancy and extra-abdominal factors, then perform targeted abdominal, groin and cardiorespiratory examination.
- Obtain focused bloods, urinalysis and pregnancy testing, provide analgesia, fasting and fluid correction, then repeat examination for evolving localisation.