01OverviewDefinition, clinical context and the essential points that orientate the chapter.
Establish pregnancy and family risk before examining. Record breech timing and mode of delivery, first-degree relatives who required a harness or operation, oligohydramnios, multiple pregnancy, torticollis and foot deformity. Confirm whether NIPE screening and any indicated ultrasound occurred and obtain the actual result rather than parental recollection of normal. Ask about swaddling and carrying position. In an older infant, ask about asymmetrical crawling, leg length, standing and delayed or unusual gait.
Examine on a warm firm surface with the infant settled and nappy removed. Inspect spontaneous movement, leg length and resting hip position. Ortolani gently abducts and lifts the proximal femur to identify a dislocated head that reduces; Barlow assesses whether a centred head can be displaced posteriorly. Repeat forceful attempts can injure the hip and add no value. Compare abduction with hips flexed, perform Galeazzi knee-height assessment and examine spine, feet and neck.
Screening timing matters. The NIPE physical examination occurs within the newborn period and at six to eight weeks. Selective ultrasound is offered for defined risk factors and abnormal examination, commonly at four to six weeks so physiological laxity can settle without delaying treatment. A normal early examination does not cancel risk-factor imaging. Conversely, a normal ultrasound does not justify ignoring later limited abduction or gait asymmetry; re-examine and refer from the current findings.
Ultrasound shows femoral-head coverage, acetabular morphology and movement before ossification obscures the view. Static Graf measurements and dynamic stability should be interpreted by a trained service within its pathway. Mild immaturity may be observed with repeat scanning, while displacement or instability requires orthopaedic review. In an older infant, AP pelvis assesses Shenton line, acetabular index and ossific nucleus position. Additional views are selected by the surgeon rather than routinely forcing a frog-leg pose.
Pavlik harness is used early when the hip is reducible and appropriate for dynamic treatment. Flexion directs the head toward the acetabulum while straps permit controlled abduction; the family should not adjust straps unless trained. Review clinically and with ultrasound soon after application and at protocolled intervals. If the hip is not reduced promptly, abandon prolonged unsuccessful harness use because pressure against the acetabular rim increases osteonecrosis and posterior-wall damage.
When harness treatment fails or presentation is later, reduce under anaesthesia. Arthrogram demonstrates the cartilaginous head, labrum and medial dye pool and tests whether a stable safe zone exists. Adductor tenotomy may permit reduction, followed by a carefully moulded hip spica. CT or MRI can confirm position according to local radiation and anaesthetic practice. Open reduction removes soft-tissue obstacles when closed reduction is not concentric or stable.
Residual structural disease is treated according to age and anatomy. Femoral shortening or derotation reduces pressure and corrects version; pelvic procedures redirect or reshape acetabular coverage. A walking child with high dislocation needs a specialist plan balancing reduction, vascular risk, stiffness and later function rather than simply applying an infant pathway late. Bilateral disease can lack obvious asymmetry, so gait, lumbar posture and pelvic imaging must be interpreted together.
Follow development beyond removal of harness or cast. Check abduction, leg length, gait, skin and femoral-nerve function and obtain radiographs until acetabular development is convincingly normal. Teach hip-safe swaddling with flexion and room for abduction. Provide urgent return advice for reduced limb movement, cast soiling or tightness, colour change, swelling, pressure injury or fever. Shared decisions should include the possibility of later osteotomy despite apparently successful early reduction.
Key points
- DDH ranges from shallow acetabulum to a dislocatable or fixed dislocated hip; early disease may be entirely painless and visually subtle.
- NIPE examines hips within 72 hours of birth and again at six to eight weeks, using Ortolani and Barlow manoeuvres plus later abduction and leg-length assessment.
- A true Ortolani clunk is the femoral head reducing over the acetabular rim; a soft click without translation is not equivalent.
- First-line imaging for a young infant is dynamic hip ultrasound, targeted at about four to six weeks for national risk factors or abnormal examination.
- After femoral-head ossification, AP pelvic radiography becomes the reference structural study for acetabular index, head position and residual dysplasia.
- First-line treatment for a stable reducible early hip is a supervised Pavlik harness holding flexion while allowing safe abduction; double nappies do not treat DDH.
- Failure of harness treatment or later fixed dislocation requires examination under anaesthesia, arthrogram and closed reduction with spica, or open reduction when concentric stability cannot be achieved.
- Continue radiographic follow-up after successful reduction because residual dysplasia can persist silently and may require femoral or pelvic osteotomy.
02AetiologyUnderlying causes, associations and risk factors, with why each one matters.
Breech mechanical position
Extended knees and constrained late-pregnancy hip position increase instability, making breech presentation a major selective-screening indication even when examination is normal.
Familial and sex susceptibility
Female sex and a first-degree relative who needed DDH treatment indicate inherited capsular and acetabular susceptibility rather than a single Mendelian disorder.
Packaging and associated conditions
First pregnancy, oligohydramnios, large size, torticollis and foot deformity reflect uterine constraint and should prompt careful hip assessment.
Postnatal positioning
Tight swaddling with hips extended and adducted can maintain displacement, whereas flexed abducted hip-safe positioning permits physiological development.
03PathophysiologyThe causal sequence from the underlying abnormality to symptoms and harm.
- 1Capsular instability
A lax capsule permits the femoral head to translate or dislocate from the acetabulum during late fetal or early postnatal life.
- 2Reciprocal growth failure
The centred femoral head stimulates acetabular depth; persistent displacement leaves a shallow roof and increasingly obstructed reduction.
- 3Soft-tissue interposition
With time, tightened adductors, pulvinar tissue, inverted labrum and hypertrophied ligamentum teres can prevent stable concentric closed reduction.
- 4Proximal-femoral remodelling
Chronic malposition alters femoral anteversion, neck shape and abductor mechanics, producing limp and increasing later reconstructive complexity.
- 5Treatment-related perfusion risk
Forced abduction or excessive pressure during reduction can compromise femoral-head blood supply and cause osteonecrosis and growth disturbance.
04Clinical features and red flagsSymptoms, examination findings, patterns of presentation and time-critical warnings.
A palpable head moving into the acetabulum during gentle abduction identifies a dislocated but reducible hip.
Posterior translation of a centred head under controlled examination indicates dislocatability and needs pathway-based imaging and review.
After early infancy, asymmetrical restriction with the hips flexed is a key sign of fixed unilateral displacement.
Unequal knee height with hips and knees flexed reflects apparent femoral shortening from unilateral proximal displacement.
Trendelenburg limp, toe walking on the short side or bilateral waddling can be the first sign after missed screening.
Reduced kicking, hip-extension weakness, skin damage or persistent sonographic displacement requires prompt device and treatment review.
05InvestigationsWhat to request, why it matters and how to interpret it.
Read from the initial assessment onwards. Tests may run in parallel in urgent care; first-line, preferred, confirmatory, definitive and gold-standard labels appear only when the chapter explicitly states them.
- 01
First-line infant hip ultrasoundFirst stepFirst line - Why
- Assess acetabular morphology, femoral-head coverage and dynamic stability before substantial ossification.
- Interpretation and limitations
- Use trained service measurements and age-adjusted classification; unstable or persistently dysplastic hips require specialist management.
- 02
AP pelvic radiograph in the older infant - Why
- Measure acetabular development and head position after ultrasound becomes less informative.
- Interpretation and limitations
- Assess acetabular index, Shenton line and ossific-nucleus position symmetrically; normal ossification varies with age.
- 03
Arthrogram during planned reduction - Why
- Display cartilaginous structures, obstacles and the stable safe zone under anaesthesia.
- Interpretation and limitations
- A wide medial dye pool or unstable eccentric position suggests interposition and may require open reduction.
- 04
Post-reduction cross-sectional imaging - Why
- Confirm concentric head position within a spica when clinical and plain assessment cannot show cartilage reliably.
- Interpretation and limitations
- Use MRI or low-dose CT according to the specialist protocol, balancing anaesthesia, radiation and diagnostic confidence.
- 05
Serial residual-dysplasia radiographs - Why
- Track acetabular growth, head development and complications after apparent successful treatment.
- Interpretation and limitations
- Persistent elevated acetabular index, subluxation or disrupted Shenton line can prompt reconstructive planning before symptoms develop.
06Differential diagnosisRealistic alternatives and the features that help distinguish them.
Physiological neonatal laxity
Mild early ultrasound immaturity may resolve as the hip matures, but clinical instability still needs protocolled surveillance rather than casual reassurance.
Neuromuscular hip displacement
Cerebral palsy, spina bifida and other muscle imbalance causes later progressive migration with a different surveillance and reconstruction pathway.
Septic arthritis
Fever, pain and pseudoparalysis indicate acute infection requiring aspiration and washout rather than screening ultrasound follow-up.
Proximal femoral deficiency
Congenital shortening and abnormal femoral anatomy creates leg-length difference without a typical reducible neonatal hip dislocation.
Normal skin-fold asymmetry
Unequal thigh creases are common and non-specific; abduction, leg length and stability findings determine further investigation.
07ManagementImmediate care, first-line treatment, alternatives and escalation.
01ScreenComplete NIPE and risk imagingFirst stepA newborn or young infant attends routine examination or has a recognised DDH risk factor.+
- 1Perform gentle bilateral stability, abduction, leg-length and associated-condition examination at the specified newborn and six-to-eight-week contacts.
- 2Arrange targeted ultrasound at the national programme interval for abnormal examination or qualifying breech or family risk.
- 3Refer a clinically dislocated hip directly rather than waiting for routine screening administration.
- 4Record completion and chase non-attendance because an unperformed scan is not a negative result.
02Early reducibleCentre the hip dynamicallyUltrasound confirms a reducible unstable or dislocated hip suitable for harness treatment.+
- 1Apply a Pavlik harness through the specialist service with hips flexed and free from forced abduction.
- 2Teach strap, skin, nappy, clothing and limb-movement care and prohibit untrained adjustment.
- 3Verify reduction promptly by ultrasound and stop an ineffective harness rather than persisting against the rim.
- 4Continue protocolled imaging after weaning to detect residual acetabular dysplasia.
03Fixed or lateObtain concentric stable reductionThe hip is irreducible, harness treatment fails or diagnosis occurs beyond the early harness window.+
- 1Plan examination under anaesthesia and arthrogram with adductor release when required.
- 2Use gentle closed reduction and hip spica only when a concentric position remains stable within a safe abduction range.
- 3Proceed to open reduction when tissue blocks or instability prevent safe closed treatment.
- 4Confirm cast position and monitor perfusion, pressure areas, hygiene and redislocation throughout spica treatment.
04Residual diseaseRestore coverage and mechanicsSerial imaging shows persistent dysplasia, subluxation or deformity after reduction.+
- 1Define acetabular orientation, femoral version, head shape, gait and leg length with age-appropriate imaging.
- 2Select femoral shortening or derotation and pelvic redirection or reshaping according to age and anatomy.
- 3Explain osteonecrosis, stiffness, redislocation and later arthritis alongside expected functional benefit.
- 4Continue growth surveillance because reconstruction changes mechanics but does not end developmental risk.
Key medicines and prescribing safety2 treatments · regimens, roles and cautions+
Paracetamol after reduction or cast application
For infants and children, prescribe the age- and weight-specific oral dose from the current BNF for Children and local postoperative protocol, documenting milligrams and maximum daily frequency rather than household spoon measures.Verify weight, formulation concentration, dosing device, liver risk and all combination products; escalating pain or reduced kicking needs examination rather than dose escalation alone.
Ibuprofen for selected postoperative children
Use an age- and weight-appropriate oral BNFC regimen for the shortest necessary period when hydration, renal function and bleeding risk are satisfactory and the operating team permits NSAID use.Avoid in dehydration, significant renal disease, active gastrointestinal bleeding, NSAID-sensitive asthma and selected postoperative settings; follow age restrictions and product concentration carefully.
08ComplicationsImportant consequences, why they occur and why they matter clinically.
Residual acetabular dysplasia
A hip can remain centred yet have inadequate roof development, requiring serial radiography and sometimes pelvic osteotomy later.
Femoral-head osteonecrosis
Excessive abduction, pressure or vascular injury during reduction can deform the head and proximal physis with lifelong consequences.
Redislocation or failed reduction
Persistent soft-tissue obstruction, poor harness fit or unstable spica position can leave the femoral head eccentrically placed.
Leg-length and gait abnormality
Untreated unilateral dislocation produces shortening and abductor limp, while bilateral disease can cause waddling and lumbar hyperlordosis.
Early osteoarthritis
Residual incongruity and concentrated edge loading damage cartilage, causing adult pain and premature arthroplasty even after childhood treatment.
09Monitoring and follow-upTreatment response, safety checks and longer-term review.
- During harness care, inspect skin, strap position, spontaneous kicking and femoral-nerve function at every review.
- Use ultrasound to document timely reduction rather than assuming a comfortable infant has a centred hip.
- In spica, check toes, swelling, colour, pressure areas, cast edges, hygiene and fit as the child grows.
- After reduction, follow acetabular index, head position and femoral-head growth for residual dysplasia and osteonecrosis.
- At walking age, record leg length, abductor strength, gait, hip range and family-reported activity.
- Track every planned image and appointment to completion because DDH can remain painless while dysplasia progresses.
10Special situationsVariants, exceptions and circumstances that change the usual approach.
A click is not a clunk
Soft-tissue clicking is common, whereas palpable translation of the femoral head carries the pathological significance.
Normal examination does not cancel risk
Qualifying breech or family history still triggers selective ultrasound because instability can be subtle or intermittent.
The head shapes the socket
Early concentric reduction restores the reciprocal growth stimulus and reduces later acetabular reconstruction.
Harness failure has a clock
Continuing an unreduced hip in flexion pressure can damage the acetabular rim and femoral-head perfusion.
Bilateral disease hides asymmetry
Equal leg lengths and skin folds can coexist with two dislocated hips, making abduction and gait pattern important.
Reduction is not the endpoint
Acetabular development can remain deficient after centring, so radiographic surveillance prevents a silent late presentation.
11Common pitfallsFrequent interpretation and management errors.
- 01
Equating an isolated hip click with a definite dislocation or dismissing a true Ortolani clunk as a click.
- 02
Cancelling risk-factor ultrasound because the newborn examination was normal.
- 03
Repeating forceful Barlow or Ortolani manoeuvres at multiple encounters instead of referring.
- 04
Using double nappies or tight swaddling as treatment for a confirmed unstable hip.
- 05
Continuing an unsuccessful Pavlik harness without prompt sonographic proof of reduction.
- 06
Discharging after reduction without long-term imaging for residual dysplasia and osteonecrosis.