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Autoimmune pancreatitis

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Escalate

Obstructive jaundice with fever or sepsis, organ-threatening extra-pancreatic disease, or a concerning pancreatic mass needs urgent specialist assessment; an empirical steroid trial must not delay cancer or infection management.

Synopsis

Distinguish the two major autoimmune pancreatitis subtypes, complete a pancreatic-cancer assessment before considering steroids, and monitor a source-based induction regimen for response, toxicity and relapse.

  • Type 1 autoimmune pancreatitis is the pancreatic manifestation of IgG4-related disease and can involve bile ducts, salivary glands, kidneys and other organs.
  • Type 2 is a distinct duct-centred disorder with granulocytic epithelial lesions; inflammatory bowel disease is an association and serum IgG4 may be normal.
  • Combine imaging, tissue, serology and other-organ assessment. Neither a raised IgG4 nor a small negative biopsy settles the diagnosis; complete the malignancy assessment before a steroid trial, whose response does not itself exclude cancer.

Key red flags

A focal pancreatic mass, progressive jaundice or unexplained weight loss without a completed diagnostic assessment.

Fever, rigors or deteriorating physiology with biliary obstruction.

New infection, severe psychological symptoms, visual change or inability to retain essential steroid treatment during therapy.

The cancer mimic

A focal mass, jaundice and weight loss can occur in either autoimmune disease or cancer. Mild IgG4 elevation and non-specific inflammatory cells are insufficient reassurance. Assess the whole imaging and pathological picture in the pancreatic multidisciplinary team; a discordant or inadequately sampled lesion may require repeat investigation before treatment is safe.

Investigation priorities

01
Pancreatic CT or MRI/MRCPFirst step

Assess the parenchymal and ductal pattern and evaluate a possible malignancy.

Management branches

Worked caseTreating confirmed type 1 disease safely

A 50 kg adult develops jaundice and mild upper abdominal discomfort. Imaging shows diffuse pancreatic enlargement and bile-duct involvement without features of cholangitis.

  1. The pancreatic team reviews imaging, markedly elevated IgG4 and a representative core biopsy showing the characteristic type 1 pattern. The complete assessment is concordant and important malignant alternatives have been addressed; the absence of malignant cells is not used in isolation. After infection, glucose, bone and medicine review, the team prescribes Milpharm prednisolone 5 mg soluble tablets, six tablets (30 mg) orally each morning for a planned four-week induction, with written safety and taper instructions.
  2. At two weeks the jaundice and discomfort have improved, bilirubin has fallen from 108 to 34 micromol/L and repeat imaging shows reduced pancreatic and biliary inflammation. Glucose and blood pressure remain acceptable and there are no significant psychological or infectious adverse effects. The team continues the induction to the planned four-week review rather than stopping on the strength of early symptom relief.

Key medicines

Milpharm prednisolone 5 mg soluble tabletsSelected specialist off-label adult AIP induction: 0.6 mg/kg orally once daily in the morning for a planned four weeks; a 50 kg adult receives 30 mg (six 5 mg tablets). Dissolve in water, or swallow the tablets whole. Review clinical, biochemical and imaging response at two to four weeks. If remission permits, an initial taper of 5 mg every two weeks is one protocol-based approach; slow reductions around 7.5 mg/day and below, guided by disease activity and adrenal recovery. Do not stop abruptly after this course.Do not use with product hypersensitivity or untreated systemic infection; specific anti-infective treatment is required when infection is present. Avoid live-virus immunisation during immunosuppression. Give steroid treatment information and instructions for illness, surgery or inability to retain tablets; prolonged therapy can suppress the adrenal axis. Monitor glucose, BP, fluid status, potassium, infection, mood, eyes and bone risk. Renal impairment/HF increases fluid-retention concern; liver failure can increase exposure and needs closer monitoring, with no fixed adjustment table. Use particular care with older age, diabetes, osteoporosis, peptic ulcer, glaucoma, severe affective illness, recent MI, TB or systemic sclerosis; the latter carries renal-crisis risk, so monitor BP and renal function. Seek urgent advice for chickenpox/shingles or measles exposure, serious infection, severe mood or suicidal symptoms; do not independently stop established steroids. Refer new visual disturbance for assessment. Avoid strong CYP3A-inhibitor combinations unless benefit justifies risk; enzyme inducers may reduce effect. Review NSAIDs/aspirin for GI bleeding, monitor INR with warfarin and potassium with potassium-lowering drugs; methotrexate may increase blood toxicity. Pregnancy requires a benefit–risk decision. Up to 40 mg/day during breastfeeding is unlikely to cause systemic infant effects; higher doses need review. Store below 30°C protected from light.
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Sources and review status6 sources · checked 8 Sept 2026 · clinical review pending
Sources

Sources and review status

National guidance is shown before implementation-dependent detail. Typical adult dose examples remain subject to patient factors, contraindications and the live BNF or specialist protocol. Source check completed 8 Sept 2026; clinical approval remains outstanding.

Authoring stateRapid draftClinical stateAwaiting reviewJurisdictionUnited Kingdom