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Educational draft · awaiting clinical reviewUse Rapid for revision, not patient-care decisions. Check current national and local guidance and the BNF or BNFC before acting.
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Post-revascularisation surveillance

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Symptoms override the calendar

New rest pain, a cold or weak limb, sensory or motor deficit, sudden abdominal or back pain, collapse, bleeding, fever with graft pain, or a rapidly expanding pulsatile mass may signal occlusion, rupture, infection or pseudoaneurysm.

Action: Do not wait for the next surveillance appointment. Arrange same-day emergency vascular assessment, compare pulses and perfusion with baseline, and obtain urgent duplex or CTA according to the suspected failure while beginning resuscitation, sepsis or limb-ischaemia care.

Synopsis

Build a repair-specific surveillance plan that detects clinical deterioration, conduit or device failure and disease progression while reinforcing secondary prevention, functional recovery and timely re-intervention.

  • New ischaemic, bleeding, infective or rupture symptoms bypass routine follow-up: arrange urgent vascular assessment and targeted duplex or CTA rather than waiting for the scheduled surveillance test.
  • After lower-limb revascularisation for intermittent claudication, combine symptoms, wound and pulse examination, ankle or toe pressures and risk-factor review; add duplex for autologous vein grafts and below-knee interventions because clinically silent stenosis can precede occlusion.
  • After standard EVAR with a device of proven durability, obtain CTA within 30 days. Low-frequency imaging during the first five years requires no endoleak or compromised seal, anatomy within the device instructions for use, adequate component overlap, at least 10 mm proximal and distal seal, neck diameter no greater than 30 mm, neck angulation no greater than 60°, iliac diameter no greater than 20 mm and no investigational or new device; non-standard and complex EVAR need individualised follow-up, and every EVAR patient needs long-term imaging regardless of initial risk.

Key red flags

Sudden loss of a previously palpable pulse, falling ankle pressure, rest pain or neurological deficit suggests graft or stent thrombosis and acute limb ischaemia.

New abdominal or back pain, hypotension, sac enlargement, a pulsatile mass or gastrointestinal bleeding after aortic repair suggests rupture, endoleak, pseudoaneurysm or fistulation.

Fever, recurrent bacteraemia, wound breakdown, perigraft pain or a draining sinus suggests graft infection and requires cultures plus urgent graft-focused imaging.

A non-healing wound, recurrent claudication or progressive tissue loss despite patent proximal reconstruction may indicate inflow, conduit, outflow or microcirculatory failure.

Infection or anastomotic failure

New fever, graft pain, wound change, pulsatile mass or bleeding requires urgent assessment for graft infection and pseudoaneurysm rather than routine patency review.

Reasoning priorities

01
Clinical review with pulse, wound and ankle or toe-pressure assessment

Establish whether perfusion, tissue healing, symptoms and cardiovascular prevention are improving from the postoperative baseline.

New rest pain, tissue loss, pulse change or reproducible pressure decline prompts anatomical imaging; incompressible ankle arteries require toe pressure or another validated perfusion measure.

Worked reasoning

Worked caseSilent stenosis after infra-inguinal vein bypass

After bypass for intermittent claudication, the operative wound is healed and walking symptoms improved, but the graft remains within the early surveillance period.

  1. Context: confirm the original indication, target vessel, conduit and postoperative baseline; ask about recurrent claudication, rest pain, wound progress and cardiovascular or bleeding events.
  2. Reasoning: examine the limb and conduit, compare ankle or toe pressure with baseline and perform duplex because a palpable pulse and symptomatic improvement do not exclude a clinically silent graft stenosis.
  3. Action: if duplex shows a haemodynamically important correctable stenosis, review inflow, outflow and technical anatomy promptly with the vascular team rather than waiting for graft thrombosis.
  4. Verification: after observation or intervention, document symptoms, wound, pressures and duplex result, then continue surveillance and secondary prevention through the period when graft failure is concentrated.
Stable EVAR pathwayEarly CTA risk stratification after standard EVAR

The patient is clinically stable after standard EVAR with a proven device, and early CTA is available to classify device, seal, endoleak, morphology and sac risk.

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Sources and review status4 sources · checked 13 Sept 2026 · clinical review pending
Sources

Sources and review status

National guidance is shown before implementation-dependent detail. Apply principles in context and verify current guidance when a decision affects care. Source check completed 13 Sept 2026; clinical approval remains outstanding.

Authoring stateRapid draftClinical stateAwaiting reviewJurisdictionUnited Kingdom