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Educational draft · awaiting clinical reviewUse Rapid for revision, not patient-care decisions. Check current national and local guidance and the BNF or BNFC before acting.
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Pulmonary embolism recognition and risk stratification

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Suspected high-risk pulmonary embolism

Shock, persistent hypotension, cardiac arrest or rapidly worsening hypoxaemia with suspected pulmonary embolism signals immediate risk of right-ventricular failure and death.

Action: Call the resuscitation and senior multidisciplinary teams, provide monitored ABC support, obtain urgent diagnostic or bedside cardiac evidence without unsafe transfer, and prepare anticoagulation and reperfusion according to bleeding risk.

Synopsis

Recognise pulmonary embolism promptly, choose the correct probability-led diagnostic branch, and distinguish physiological severity from suitability for carefully supported outpatient care.

  • Assess haemodynamic stability before calculating any diagnostic or outpatient score: shock or persistent hypotension places the patient in the emergency high-risk branch.
  • In non-pregnant adults with low overall clinical suspicion and a feasible alternative diagnosis, consider PERC; otherwise use two-level Wells: above 4 needs immediate CTPA, while 4 or less needs D-dimer, with interim therapeutic anticoagulation whenever the indicated test is delayed.
  • Only physiologically low-risk patients should enter outpatient assessment, using a validated prognostic tool plus bleeding, comorbidity, social and follow-up checks.

Key red flags

Systolic hypotension, obstructive shock, syncope with ongoing compromise, or cardiac arrest requires an emergency PE pathway.

Severe or escalating hypoxaemia, altered consciousness, cold peripheries, oliguria or rising lactate indicates impaired right-heart output.

Active major bleeding, recent intracranial disease or recent major surgery materially changes anticoagulation and reperfusion choices.

Pregnancy, severe renal impairment or contrast allergy changes imaging selection but must not permit diagnostic drift or dangerous delay.

Haemodynamic instability

Persistent hypotension, shock, cardiac arrest, cool peripheries, confusion, oliguria or elevated lactate reflects inadequate output from acute right-ventricular pressure overload and mandates emergency escalation.

Investigation priorities

01
Two-level PE Wells score and D-dimerFirst step

Select the NICE pre-test probability branch in non-pregnant adults before definitive thoracic imaging.

Management branches

Emergency pathwayUnstable suspected or confirmed PE

Shock, persistent hypotension, cardiac arrest or rapidly worsening organ perfusion is present.

  1. Start monitored ABC resuscitation, summon senior critical-care and PE expertise, establish vascular access and avoid excessive fluid loading that may worsen right-ventricular distension.
  2. Obtain the fastest safe confirmation and right-heart assessment, begin continuous UFH for confirmed unstable PE when not contraindicated, and assess systemic thrombolysis immediately against bleeding hazards.
Diagnostic pathwayStable non-pregnant adult with suspected PE

PE is clinically suspected in a non-pregnant adult without haemodynamic instability.

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Sources and review status4 sources · checked 13 Sept 2026 · clinical review pending
Sources

Sources and review status

National guidance is shown before implementation-dependent detail. Apply principles in context and verify current guidance when a decision affects care. Source check completed 13 Sept 2026; clinical approval remains outstanding.

Authoring stateRapid draftClinical stateAwaiting reviewJurisdictionUnited Kingdom