Doctor’s Passport

Find your next topic

Explore the current textbook

Available drafts · Clinical review pending
Membership
Educational draft · awaiting clinical reviewUse Rapid for revision, not patient-care decisions. Check current national and local guidance and the BNF or BNFC before acting.
Rapid

Liver, pancreas and renal lesion characterisation

Essential points for quick revision.

Saved on this device
!
Complicated focal lesion

Haemorrhage, rupture, biliary or urinary obstruction, pancreatitis, sepsis or vascular invasion may convert an incidental lesion into urgent disease.

Action: Stabilise, identify the complication with an appropriate phase or modality and involve hepatobiliary, pancreatic, urological or interventional specialists promptly.

Synopsis

Characterise focal liver, pancreatic and renal lesions with organ-specific multiphasic imaging, separate simple benign appearances from indeterminate or malignant features, and direct each lesion to the correct specialist pathway.

  • Characterisation asks whether a lesion contains fat, fluid, blood or enhancing tissue and how it behaves across phases; size alone rarely answers this.
  • First identify organ, patient cancer and liver-risk context, symptoms, prior imaging and whether the study was adequately protocoled.
  • Use contrast-enhanced MRI preferentially when a benign liver lesion is suspected, while CT provides rapid assessment when malignancy or complications are concerns.

Key red flags

Hypotension, falling haemoglobin or severe pain with a liver or renal mass suggests haemorrhage and requires urgent multiphase assessment.

Obstructive jaundice, pancreatic-duct dilatation, upstream atrophy or a solid pancreatic component requires expedited specialist evaluation.

Enhancing soft tissue in a cystic renal lesion, renal-vein extension or metastatic features raises RCC concern and needs uro-oncology review.

A new liver lesion in cirrhosis or known cancer follows a high-risk pathway rather than a generic incidental-lesion algorithm.

Pancreatic warning

Solid component, mural nodule, main-duct change, obstructive jaundice or upstream atrophy raises concern for malignant or pre-malignant disease.

Renal invasion

Renal sinus or perinephric extension, renal-vein thrombus, nodes and distant disease affect staging and treatment planning.

Lesion complication

Rupture, haemorrhage, infection, duct obstruction or organ dysfunction requires acute assessment separate from long-term neoplastic risk.

Reasoning priorities

01
Multiphasic liver CT or MRI

Assess arterial, portal and delayed enhancement and background liver.

A characteristic pattern may establish diagnosis; indeterminate lesions in high-risk liver require specialist pathways rather than generic follow-up.

Worked reasoning

Worked caseIndeterminate portal-venous lesion

A routine CT identifies an unexpected focal lesion that lacks a definitive benign pattern.

  1. Context: establish organ, symptoms, liver disease, cancer history, renal function, pancreatitis and hereditary risk.
  2. Reasoning: inspect whether fluid, fat, blood or enhancement is present and whether the original phase can answer the organ-specific question.
  3. Outcome: choose dedicated liver MRI, pancreatic CT or MRI-MRCP, or multiphasic renal CT or MRI and route to the relevant MDT if suspicious.
  4. Verification: compare prior images, document the final classification and ensure any surveillance or intervention has a named owner.
Open full textbook Answer 2 questions
Sources and review status3 sources · checked 13 Sept 2026 · clinical review pending
Sources

Sources and review status

National guidance is shown before implementation-dependent detail. Apply principles in context and verify current guidance when a decision affects care. Source check completed 13 Sept 2026; clinical approval remains outstanding.

Authoring stateRapid draftClinical stateAwaiting reviewJurisdictionUnited Kingdom